First P2 in #Thailand Oxford Nanopore #Siriraj ✨ #Bangkok

First #acmg24 ! Learn a lot from geneticists and scientists. Good to see my conference buddy from ClinGen Alicia Byrne - Systematic review of kidney panel diagnostic yield show 40% positive rate !! - In UDN, adult with kidney disease has the highest diagnostic yield

🌟Check out the March Issue of KI! 🌟 Featured topics include: 🔹Fatty livers and #CKD 🔹A #proteomic atlas of #kidney #amyloid plaques 🔹Genetics of idiopathic #collapsingglomerulopathy 🔹Anti-#nephrin antibodies in recurrent #FSGS 👉doi.org/10.1016/j.kint…

Amazing !!!

With tremendous joy, I share that our patient who received a gene-edited pig kidney transplant was discharged home today after removal of his dialysis access. We hope that this is a first step that will bring hope to many patients waiting for a kidney transplant #xenotransplant

Check out our review on "SGLT2i and GU infections" in JACC Journals 🔗: shorturl.at/gsGL7 Here we discuss - SGLT2i mechanisms/benefits - Trial/real-world evidence of SGLT2i/GU ifx - Risk factors - Practice considerations 🧵 to follow (1/5)

Eduardo Slatopolsky, MD, will be missed by many, both here at WU in STL Nephrology and throughout the world. He leaves behind a legacy of groundbreaking contributions to our field and had a profound impact on the lives of countless patients and colleagues. nephrology.wustl.edu/obituary-dr-ed…

8 years ago, he had made me felt so welcome after moving to a new city and still so new to the US. Little that I know at that time how much he has contributed to the field of kidney medicine. Thank you for your inspiration WU in STL Nephrology Ben Humphreys

Exiciting to see the opportunity to see what we were not able to see from #genomicthailand A log of kidney patients to be looked at

This is amazing Fouad Chebib, MD, FASN ⭐️👌#nephtwitter #medtwitter

#ERA24 and International Society of Glomerular Disease highlight! Tobi Huber showing his lab’s incredible results expanding anti-nephrin antibody studies in diffuse podocytopathies and proving pathogenicity in the mouse. Proud to have sparked the field and inspired this breakthrough. #newideasnewtherapiesforNS

Preprint: Genotype-first analysis in an unselected health system-based population reveals variable phenotypic severity of COL4A5 variants medrxiv.org/content/10.110…

I could laugh along with Sophia Ambruso Nayan Arora,MD AC Gomez Swapnil Hiremath @[email protected] Joel M. Topf, MD FACP for an hour (and there’s probably that much here), but the picks are great—even the practice points! These aren’t your parents’ ace inhibitor, bicarb, and low protein guidelines.

Contrary to expectations: Higher protein intake is linked to lower mortality in adults with chronic kidney disease according to a large new study. Finally, we have a study. It's time to abandon the low-protein approach. Link to study: jamanetwork.com/journals/jaman…

Is kidney biopsy the only way to diagnose immune checkpoint inhibitor-associated acute kidney injury (ICI-AKI)? Maybe not. In a new paper in Journal of Clinical Investigation , David Leaf and I report that PET-CT may have a role. A brief #tweetorial #Thread (jci.org/articles/view/…).

Developing understanding of Alport Syndrome: Kandai Nozu provided an overview of Alport Syndrome in Asia, Judy Savige updated on the diversity of clinical features, and Rachel Lennon explained what happens in the kidneys in Alport Syndrome. #AlportWorkshop

Thank you for the opportunity to share about VUS(es) in #alportworkshop With Alport syndrome alliance Alport UK Excited to bring these testing to Thailand in the upcoming year

Thank you for sharing ! Alport UK #alportworkshop

Honored to be invited to #precise2024 #ihcc to present my experience in using what I learned from The Brigham Nephrology @harvardmed and Matt Sampson to Thai genetic kidney disease. My heart rate pre-talk is in the 100s ! Thanks to Dr. Kar Hui for the nomination

For Kidney Health Advocates- lets all get on the same page: 1) only <5% of incident CKD is caused by HTN;2) progression of CKD can be slowed by treatment of HTN when proteinuria is concomitantly present; 3) HTN is mostly consequent to CKD, not caused by CKD.